Effectiveness of biomedical risk assessment as an aid for smoking cessation: a systematic review.

TitleEffectiveness of biomedical risk assessment as an aid for smoking cessation: a systematic review.
Publication TypeJournal Article
Year of Publication2007
AuthorsBize, R, Burnand, B, Mueller, YK, Cornuz, J
JournalTobacco Control
Volume16
Issue3
Pagination151-6
Date Published2007 Jun
DOI10.1136/tc.2006.017731
ISSN1468-3318
KeywordsBiofeedback, Psychology, Female, Genetic Predisposition to Disease, Humans, Male, Randomized Controlled Trials as Topic, Risk Assessment, Smoking, Smoking Cessation, Treatment Outcome
Abstract

OBJECTIVE: To determine the efficacy of biomedical risk assessment (eg, exhaled carbon monoxide (CO), or genetic susceptibility to lung cancer) as an aid for smoking cessation.

DATA SOURCES: Cochrane Tobacco Addiction Group Specialized Register, Cochrane Central Register of Controlled Trials, Medline (1966-2004) and EMBASE (1980-2004).

STUDY SELECTION: Randomised controlled smoking cessation interventions using biomedical tests with at least 6 months follow-up.

DATA EXTRACTION: Two reviewers independently screened all search results (titles and abstracts) for possible inclusion. Each reviewer then extracted data from the selected studies, and assessed their methodological quality based on the CONSORT (Consolidated Standards of Reporting Trials) statement criteria.

DATA SYNTHESIS: Of 4049 retrieved references, eight trials were retained for data extraction and analysis. Three trials isolated the effect of exhaled CO on smoking cessation rates resulting in the following ORs and 95% CIs: 0.73 (0.38 to 1.39), 0.93 (0.62 to 1.41) and 1.18 (0.84 to 1.64). Measurement of exhaled CO and spirometry were used together in three trials, resulting in the following ORs (95% CI): 0.60 (0.25 to 1.46), 2.45 (0.73 to 8.25) and 3.50 (0.88 to 13.92). Spirometry results alone were used in one other trial with an OR (95% CI) of 1.21 (0.60 to 2.42). Ultrasonography of carotid and femoral arteries performed on light smokers gave an OR (95% CI) of 3.15 (1.06 to 9.31).

CONCLUSIONS: Scarcity and limited quality of the current evidence does not support the hypothesis that biomedical risk assessment increases smoking cessation as compared with the standard treatment.

Alternate URL

http://www.ncbi.nlm.nih.gov/pubmed/17565124?dopt=Abstract

WOS ID (UT)

000247204900003

Alternate JournalTob Control
Citation Key / SERVAL ID2517
Peer reviewRefereed
PubMed ID17565124
PubMed Central IDPMC2598501

                         

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